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RERAs Examples and APAP response
#1
RERAs Examples and APAP response
RERAs:  Many nights OCSAR shows that I have 4-8 RERAs. Each RERA shows a Flow Limit response, and I  have a corresponding APAP pressure increase response. The responses appear to be more direct and significant than those for Obstructive Apneas, Hypopneas or CAs, though those are more numerous and have greater count toward the AHI score.  Please explain the RERAs and their examples and how to address these with the APAP/CPAP settings or other ways to respond. Thanks.
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#2
RE: RERAs Examples and APAP response
RERAs can be conceptualized as sub-hypopnea events. Like the other obstructive events, the machine has a built-in response for dealing with them. Higher pressure or pressure support is the answer, if the conversation is restricted to PAP.
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#3
RE: RERAs Examples and APAP response
Thanks for the thoughtful response. When look at my OSCAR results, I see that almost all the APAP reactions are triggered by the RERAs, not the Hypopneas, and that the RERAs, in general, show much greater Flow Restrictions than the Hypopneas. It appears that the Flow restrictions are the trigger for the APAP pressure increases. See attachment for an example. 

So, coming back to my original question, what are these RERAs that often  have greater impact in Flow Restriction and Hypopneas and even some of  the Obstructive Apneas?  Example:  When I am awake, I am prone to just comfortably stopping breathing for a moment, many times not as CAs, but stopping with my lower throat.  

Again, Thanks


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#4
RE: RERAs Examples and APAP response
You have constant high flow limitation. Have you tried EPR=3?
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#5
RE: RERAs Examples and APAP response
Thanks again.  I had the EPR at 3, but lowered it to 1 or 0 to reduce the volume of CAs. While CAs do not show as flow limitations, they do affect AHI /awakening,  just as do  hypopneas. I will look at that trade off again. Other suggestions?  In general I sleep and record well for 3.5 -5 hours, but after bathroom break seem to be in half sleep with increased CAs and flow limitations for the remaining 3+ hours ( avg 8 hrs).  None of this seems to affect the consistently high O2 levels and steady HR, though latter is with leadless pacemaker.  I am an nationally competitive swimmer( by age) in good health with zero breathing problems but some moderate sleep Apnea despite CPAP/APAP and various neck and mouth devices.
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